Title: Nicotine poisoning from liquid nicotine ingestion<br/>Author: Hong Kim<br/><a href='http://umem.org/profiles/faculty/526/'>[Click to email author]</a><hr/><p>
Electronic cigarettes have been gaining popularity in the U.S. as a smokeless delivery system for nicotine. These devices require liquid nicotine (e-liquid) that are vaporized and inhaled (vaping).</p>
<p>
</p>
<p>
E-liquid can have nicotine concentration as high as 100 mg/mL, which are diluted prior to use. When ingested in high concentration and in sufficient volume (1 vial = 15 mL) patients can develop significant nicotinic toxicity. Recently a case of cardiac arrest has been reported after ingesting two 15 ml vial (100 mg/mL).</p>
<p>
</p>
<p>
Nicotine mimics the effects of acetylcholine (Ach) release by binding to nicotinic receptors located in:</p>
<ul>
<li>
Brain</li>
<li>
Spinal cord</li>
<li>
Autonomic ganglia</li>
<li>
Adrenal medulla</li>
<li>
Neuromuscular junction</li>
<li>
Chemoreceptors of carotid/aortic bodies</li>
</ul>
<p>
</p>
<p>
Clinical manifestation of toxicity (similar to cholinergic toxidrome) is biphasic with early central stimulation followed by depression. (see table below)</p>
<table align="left" border="1" cellpadding="0" cellspacing="0">
<tbody>
<tr>
<td style="width:109px;">
<p>
</p>
</td>
<td style="width:114px;">
<p>
<strong>GI</strong></p>
</td>
<td style="width:125px;">
<p>
<strong>Respiratory</strong></p>
</td>
<td style="width:126px;">
<p>
<strong>Cardiovascular</strong></p>
</td>
<td style="width:117px;">
<p>
<strong>Neurologic</strong></p>
</td>
</tr>
<tr>
<td style="width:109px;">
<p>
<strong>Early (1 hr)</strong></p>
</td>
<td style="width:114px;">
<p>
Nausea</p>
<p>
Vomiting</p>
<p>
Salivation</p>
<p>
Abdominal pain</p>
</td>
<td style="width:125px;">
<p>
Bronchorrhea</p>
<p>
Hyperpnea</p>
</td>
<td style="width:126px;">
<p>
Hypertension</p>
<p>
Tachycardia</p>
<p>
Pallor</p>
</td>
<td style="width:117px;">
<p>
Agitation</p>
<p>
Anxiety</p>
<p>
Dizziness</p>
<p>
Blurred vision</p>
<p>
Headache</p>
<p>
Hyperactivity</p>
<p>
Tremors</p>
<p>
Fasciculation</p>
<p>
Seizures</p>
</td>
</tr>
<tr>
<td style="width:109px;">
<p>
<strong>Late</strong></p>
<p>
<strong>(0.5-4 hr)</strong></p>
</td>
<td style="width:114px;">
<p>
Diarrhea</p>
</td>
<td style="width:125px;">
<p>
Hypoventilation</p>
<p>
Apnea</p>
</td>
<td style="width:126px;">
<p>
Bradycardia</p>
<p>
Hypotension</p>
<p>
Dysrhythmias</p>
<p>
Shock</p>
</td>
<td style="width:117px;">
<p>
Lethargy</p>
<p>
Weakness</p>
<p>
Paralysis</p>
</td>
</tr>
</tbody>
</table>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
</p>
<p>
<strong>Management:</strong> There is no specific antidote or reversal agent. The management of nicotine toxicity focuses on organ-specific dysfunction. </p>
<div>
e.g. bronchorrhea = atropine; apnea = intubation; seizure = benzodiazepine.</div>
<fieldset><legend>References</legend>
<ol>
<li>
Chen BC et al. Death following intentional ingestion of e-liquid. Clin Toxicol 2015;53:914-916.</li>
<li>
Kim JW et al. Liquid nicotine toxicity. Pediatr Emer Care 2015;31:517-524.</li>
</ol>
</fieldset>