Title: Hyperoxia in the Post-Arrest Patient<br/>Author: Mike Winters<br/><a href='http://umem.org/profiles/faculty/141/'>[Click to email author]</a><hr/><p>
<strong><u>Hyperoxia and the Post-Arrest Patient</u></strong></p>
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Current post-arrest guideilnes recommend titrating supplemental O2 to avoid hypoxia and limit exposure to hyperoxia.</li>
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Importantly, these recommendations are based primarily on retrospective studies that have used ABG values within the first 24 hours following ROSC.</li>
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The latest study to evaluate the impact of hyperoxia following cardiac arrest was just published in <em>Circulation</em>. </li>
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This study is a prospective, cohort study that evaluated the association between <u>early</u> hyperoxia and poor neurologic outcome in adults following cardiac arrest. (ABGs were obtained at 1 hour and 6 hours following ROSC)</li>
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Of 280 patients, 38% were exposed to early hyperoxia (defined as a PaO2 > 300 mm Hg)</li>
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<strong>Take Home Points</strong>
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<strong>Early hyperoxia was found to be an independent predictor of poor neurologic outcome at hospital discharge.</strong></li>
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<b>One hour longer duration of hyperoxia was associated with a 3% increase in the risk of poor neurologic outcome</b></li>
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<b>SaO2 could not reliably exclude the presence of hyperoxia.</b></li>
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<fieldset><legend>References</legend>
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Roberts BW, et al. Association between early hyperoxia exposure after resuscitation from cardiac arrest and neurological disability: a prospective multi-center protocol-directed cohort study. <em>Circulation</em> 2018; epub ahead of print.</p>
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